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  <front>
    <journal-meta>
      <journal-id journal-id-type="publisher">JOHS</journal-id>
      <journal-id journal-id-type="nlm-ta">Journ of Health Scien</journal-id>
      <journal-title-group>
        <journal-title>Journal of HealthCare Sciences</journal-title>
        <abbrev-journal-title abbrev-type="pubmed">Journ of Health Scien</abbrev-journal-title>
      </journal-title-group>
      <issn pub-type="ppub">2231-2196</issn>
      <issn pub-type="opub">0975-5241</issn>
      <publisher>
        <publisher-name>Radiance Research Academy</publisher-name>
      </publisher>
    </journal-meta>
    <article-meta>
      <article-id pub-id-type="publisher-id">507</article-id>
      <article-id pub-id-type="doi">http://dx.doi.org/10.52533/JOHS.2026.60112</article-id>
      <article-id pub-id-type="doi-url"/>
      <article-categories>
        <subj-group subj-group-type="heading">
          <subject>Family Medicine</subject>
        </subj-group>
      </article-categories>
      <title-group>
        <article-title>Assessment and Management of Cardiac Arrhythmias in Patients With Electrolyte Imbalances&#13;
</article-title>
      </title-group>
      <contrib-group>
        <contrib contrib-type="author">
          <name>
            <surname>Khatib</surname>
            <given-names>Arwa Farouk</given-names>
          </name>
        </contrib>
        <contrib contrib-type="author">
          <name>
            <surname>Aldawsari</surname>
            <given-names>Nasser Ibrahim</given-names>
          </name>
        </contrib>
        <contrib contrib-type="author">
          <name>
            <surname>Alshaer</surname>
            <given-names>Ahmed Abduallah</given-names>
          </name>
        </contrib>
        <contrib contrib-type="author">
          <name>
            <surname>Alkhalaf</surname>
            <given-names>Hussain Ali</given-names>
          </name>
        </contrib>
        <contrib contrib-type="author">
          <name>
            <surname>Jassas</surname>
            <given-names>Burair Mansour Al</given-names>
          </name>
        </contrib>
        <contrib contrib-type="author">
          <name>
            <surname>Ghazwi</surname>
            <given-names>Zahra Ali</given-names>
          </name>
        </contrib>
        <contrib contrib-type="author">
          <name>
            <surname>Saleh</surname>
            <given-names>Lameer Adel</given-names>
          </name>
        </contrib>
        <contrib contrib-type="author">
          <name>
            <surname>Basowad</surname>
            <given-names>Jumana Ali</given-names>
          </name>
        </contrib>
      </contrib-group>
      <pub-date pub-type="ppub">
        <day>22</day>
        <month>01</month>
        <year>2026</year>
      </pub-date>
      <volume>6</volume>
      <issue>1</issue>
      <fpage>88</fpage>
      <lpage>93</lpage>
      <permissions>
        <copyright-statement>This article is copyright of Popeye Publishing, 2009</copyright-statement>
        <copyright-year>2009</copyright-year>
        <license license-type="open-access" href="http://creativecommons.org/licenses/by/4.0/">
          <license-p>This is an open-access article distributed under the terms of the Creative Commons Attribution (CC BY 4.0) Licence. You may share and adapt the material, but must give appropriate credit to the source, provide a link to the licence, and indicate if changes were made.</license-p>
        </license>
      </permissions>
      <abstract>
        <p>Electrolyte imbalances are a frequent yet under-recognized cause of cardiac arrhythmias across a wide range of clinical settings. Alterations in serum potassium, calcium, magnesium, and sodium levels can directly affect myocardial excitability, conduction, and repolarization, often resulting in rhythm disturbances that vary in severity from mild ectopy to life-threatening ventricular tachyarrhythmias. The mechanisms underlying these arrhythmias include abnormal automaticity, triggered activity, and impaired conduction pathways, all of which are influenced by the concentration gradients and cellular handling of key electrolytes. Electrocardiographic changes often serve as early indicators of these disturbances, but interpretation must be guided by clinical context, coexisting conditions, and concurrent medications that may amplify or mask their effects. Diagnosis relies on an integrated assessment combining laboratory values with electrocardiogram monitoring to detect characteristic patterns such as prolonged QT intervals, U waves, or widened QRS complexes. In critically ill patients or those receiving therapies that disrupt electrolyte balance, continuous surveillance is essential to catch early arrhythmic manifestations before progression. Management strategies are condition-specific and must be delivered carefully to avoid overcorrection, which itself poses arrhythmic risk. Potassium and magnesium repletion, calcium stabilization, and sodium adjustments must be tailored to both the severity of derangement and the individual patient’s cardiac profile. Risk evaluation tools, combined with telemetry and serial assessments, aid in preventing recurrence and guide long-term care in patients prone to recurrent imbalances.&#13;
</p>
      </abstract>
      <kwd-group>
        <kwd>electrolyte imbalance</kwd>
        <kwd> cardiac arrhythmia</kwd>
        <kwd> ECG monitoring</kwd>
        <kwd> ion channel dysfunction</kwd>
        <kwd> risk stratification</kwd>
      </kwd-group>
    </article-meta>
  </front>
</article>